Voltage-gated Ca2+ Channels: Pharmacology, Modulation and their Role in Human Disease

Author:   Jörg Striessnig
Publisher:   Springer International Publishing AG
Edition:   2023 ed.
Volume:   279
ISBN:  

9783031384363


Pages:   316
Publication Date:   01 August 2023
Format:   Hardback
Availability:   Manufactured on demand   Availability explained
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Voltage-gated Ca2+ Channels: Pharmacology, Modulation and their Role in Human Disease


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Author:   Jörg Striessnig
Publisher:   Springer International Publishing AG
Imprint:   Springer International Publishing AG
Edition:   2023 ed.
Volume:   279
Weight:   0.718kg
ISBN:  

9783031384363


ISBN 10:   3031384369
Pages:   316
Publication Date:   01 August 2023
Audience:   Professional and scholarly ,  Professional & Vocational
Format:   Hardback
Publisher's Status:   Active
Availability:   Manufactured on demand   Availability explained
We will order this item for you from a manufactured on demand supplier.

Table of Contents

Preface.- Part 1 Modulation.-  Cav1.1Calcium Channel Signaling Complexes in Excitation–Contraction Coupling: Insights from Channelopathies.- Mechanisms of Vascular CaV1.2 Channel Regulation During Diabetic Hyperglycemia.- Sympathetic Nervous System Regulation of Cardiac Calcium Channels.- Modulation of CaV1.2 Channel Function by Interacting Proteins and Post-Translational Modifications: Implications in Cardiovascular Diseases and COVID-19.- Part 2 Pharmacological Approaches.- Pathophysiological Roles of Auxiliary Calcium Channel α2δ Subunits.- Design and Applications of Genetically-Encoded Voltage-Dependent Calcium Channel Inhibitors.- Part 3 Disease insight from channelopathies: consequences for pharmacology and therapeutic strategies.-    CACNA1C-Related Channelopathies.- CACNA1D-Related Channelopathies: From Hypertension to Autism.- CACNA1A-Related Channelopathies: Clinical Manifestations and Treatment Options.- CaV3.2 (CACNA1H) in Primary Aldosteronism,- CaV3.3 Channelopathies.- Pathophysiological Roles of Auxiliary Calcium Channel α2δ Subunits.

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