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This historic book may have numerous typos and missing text. Purchasers can download a free scanned copy of the original book (without typos) from the publisher. Not indexed. Not illustrated. 1922 Excerpt: ...basis, it occurred to me to go one step further back and combine them under the headings of the primary germ layers from which the several organs developed embryologically. To do this was a task of considerable difficulty. It raised intricate, and in some TABLE 12 Showing the relative influence of the primary germ layers in human mortality (Items 64 and 65 charged to ectoderm) TABLE 13 Shouting the relative influence of the primary germ layers in human mortality (Items 64 and 65 charged to mesoderm) cases still unsettled, questions of embryology. Furthermore, the original statistical rubrics under which the data are compiled by registrars of vital statistics were never planned with such an object as this in mind. Still the thing seemed worth trying because of the biological interest which would attach to the result, even though it were somewhat crude and, in respect of minor and insignificant details, open to criticism. It is not possible here to go into details as to how the causes of death were combined in ENDODERM Fig. 38.--Diagram showing the percentages of biologically classifiable human mortality resulting from breakdown of organs developing from the different germ layers. Upper bar of pair gives upper limit of mortality chargeable to ectoderm: lower bar gives lower limit of mortality chargeable to ectoderm. making up the final tables. For these details one must refer to the original papers. Tables 12 and 13, and Figure 38, give the results for the crude mortality of the U. S. Registration Area, England and Wales, and Sao Paulo, Brazil. The figures show that in man, the highest product of organic evolution, about 57 per cent. of all the biologically classifiable deaths result from a breakdown and failure further to function of organs arising from the ...

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