Energetics of Secretion Responses: Volume I
Author:   J.W.N. Akkerman (University Hospital/Utrecht)
Publisher:   Taylor & Francis Ltd
ISBN:  

9781138558687


Pages:   158
Publication Date:   28 January 2019
Format:   Paperback
Availability:   In Print   Availability explained
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Energetics of Secretion Responses: Volume I


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Author:   J.W.N. Akkerman (University Hospital/Utrecht)
Publisher:   Taylor & Francis Ltd
Imprint:   CRC Press
Weight:   0.453kg
ISBN:  

9781138558687


ISBN 10:   1138558680
Pages:   158
Publication Date:   28 January 2019
Audience:   Professional and scholarly ,  Professional & Vocational
Format:   Paperback
Publisher's Status:   Active
Availability:   In Print   Availability explained
This item will be ordered in for you from one of our suppliers. Upon receipt, we will promptly dispatch it out to you. For in store availability, please contact us.

Table of Contents

1. Introduction 2. Cellular Energetics 3. PreStimulation Events: Biosynthesis and Storage of Secretory Proteins 4. Transport and Storage of Biogenic Amines in Adrenal Chromaffin Granules 5. Maintenance of Responsivity 6. Energy-Dependent Steps in Signal Processing: Ligand-Induced Surface Receptor Movement and Transmembrane Interactions 7. The Role of Membrane Potentials and of Intracellular pH in Secretory Cell Stimulation 8. Phosphoinositide Metabolism. Index

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Author Information

Jan-Willem N. Akkerman PhD is the Director of the Laboratory of Haematology in the Department of Haematology at the University of Utrecht, The Netherlands. He received his Ph.D. degree in 1975 on a thesis entitled ""Human Platelet Glycolysis"" and was a visiting staff member at the Thrombosis Research Center at Temple University, Philadelphia, Pa. In 1978. His research efforts are in the field of thrombosis and atherosclerosis, with special emphasis on the role of blood platelets and the mechanisms that make these cells responsive to platelet-activating agents. His current research interests are the energetics of platelet functions, mechanisms for signal transduction, and abnormal platelet behaviour in patients with bleeding disorders or hyperaggregability.

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