Glutamate: Functions, Regulation & Disorders

Author:   Golda Chayat ,  Avital Yedidya
Publisher:   Nova Science Publishers Inc
ISBN:  

9781619425453


Publication Date:   15 June 2012
Format:   Hardback
Availability:   In stock   Availability explained
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Glutamate: Functions, Regulation & Disorders


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Overview

Glutamate is purported to be the most important transmitter for normal brain function. Nearly all excitatory neurons in the central nervous system are glutamatergic, and it is estimated that over half of all brain synapses release this agent. In this book, the authors present current research in the study of the functions, regulation and disorders related to glutamate. Topics include glutamate-scavenging in the treatment of acute brain insults; the physiology of glutamate receptors in schizophrenia; and, how glutamate works as a neurotransmitter and the role of supraspinal GABA and glutamate in the mediation of pain.

Full Product Details

Author:   Golda Chayat ,  Avital Yedidya
Publisher:   Nova Science Publishers Inc
Imprint:   Nova Science Publishers Inc
Dimensions:   Width: 26.00cm , Height: 1.80cm , Length: 18.00cm
Weight:   0.576kg
ISBN:  

9781619425453


ISBN 10:   1619425459
Publication Date:   15 June 2012
Audience:   College/higher education ,  Professional and scholarly ,  Postgraduate, Research & Scholarly ,  Professional & Vocational
Format:   Hardback
Publisher's Status:   Active
Availability:   In stock   Availability explained
We have confirmation that this item is in stock with the supplier. It will be ordered in for you and dispatched immediately.

Table of Contents

Preface; Blood Glutamate Scavenging: Insight into Neuroprotection; Glutamate & Schizophrenia; Extracellular Osmolarity Modulates Glutamate Uptake & Release in Neurons, Astrocytes & Synaptosomes; Glutamate: A Global Regulator; Calcium-Calmodulin Kinase Type II-Mediated Glutamatergic Plasticity Underlies Expression of Benzodiazepine-Withdrawal Anxiety; The Role of Supraspinal GABA & Glutamate in the Mediation & Modulation of Pain; Glutamate Receptors of the Kainate Type: An Ion Channel Becoming a Metabotropic Receptor; Index.

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